Turnover of C99 is controlled by a crosstalk between ERAD and ubiquitin-independent lysosomal degradation in human neuroglioma cells. PLoS One Dec 20;8(12):e83096.
Authors
Bustamante H.A., Rivera-Dictter A., Cavieres V.A., Muñoz V., González A., Lin Y., Mardones G.A., and Burgos P.V.* (2013)